What are the symptoms of Addison's disease in pet cats? Diagnosis and treatment of Addison's disease symptoms! Adrenal cortex hypofunction, referred to as Addison's disease, is a series of clinical syndromes caused by glucocorticoid defi...
What are the symptoms of Addison's disease in pet cats? Diagnosis and treatment of Addison's disease symptoms! Adrenal cortex hypofunction, referred to as Addison's disease, is a series of clinical syndromes caused by glucocorticoid deficiency or mineralocorticoid deficiency. Addison's disease in animals is a defect of corticosteroid hormone. This is an unusual discovery because of the shortcomings in this regard, unless it is the destruction of adrenal balance by taking medications, but fortunately, the disease can be controlled by corticosteroids, even if the cause of the defect is unknown.
For cats, adrenal hypofunction is relatively rare. No relevant reports have been found in China, and no more than 40 cases have been reported abroad. But even so, we cannot take it lightly, and we still need to have a basic understanding of it.
1. Symptoms
Adrenal cortex hypofunction in cats is usually primary, with unknown causes of the disease, and can be seen in cats aged 1-14 (average age of 4). The most common clinical symptoms are depression, dehydration, weakness and hypothermia; depression, bradycardia and abdominal pain are rare.
2. Diagnosis of
"Adison's" disease may occur in different forms. The inability to maintain normal blood sugar levels (which eventually manifests as dysfunction) may be a strong hint of insulin-secreting pancreatic tumors, and before a major abdominal surgery, it is an important test for Addison's disease.
Routine blood tests may reveal lymphocytosis and eosinophilia, as well as mild orthocytic non-regenerative anemia, and most cats with primary adrenal hypofunction experience typical hyponatremia, hypochloremia, and hyperkalemia (sodium-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-to-potassium-
III. The most important aspect of treating adrenal cortical insufficiency is the replacement of lost mineral corticosteroids. One of the methods is to take Florinef orally. Flucocortisone is usually twice a day, and the dose depends on the patient's sodium and potassium blood test. First, these electrolytes are monitored weekly. When the concentration is stable, these blood tests will be repeated 2-4 times a year. Often over time, it will be found that in order to control Addison's disease, the dose of fluorocortisone will gradually increase. Unfortunately, this medicine is relatively expensive, and since fluorocortisone has the effects of glucocorticoids, just like the mineral corticosteroids, there is no need to treat it with other drugs.
Another way to treat this condition is to inject a drug called "DOCP" (the product name is "Percorten-V"). This treatment is approximately every 25 days. First, the electrolyte is measured before injection, and the test process is usually gradually decreasing 1-2 times a year. Some neurologists also believe that DOCP can produce regulatory electrolytes better than oral fluorocortisone.
